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CASODEX is a racemate with its antiandrogenic activity being almost exclusively exhibited by the R-enantiomer of bicalutamide; the S-enantiomer is essentially inactive.

Use bicalutamide with caution in patients with hepatic impairment.

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The SERM tamoxifen has been evaluated in the treatment of gynecomastia.

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Radical prostatectomy versus expectant treatment for early carcinoma of the prostate.

Data was then analyzed for surgical complications, wound complications, and medical complications within 30 days of surgery on 4545 patients.

Here, we report the development and characterization of a novel androgen-dependent prostate cancer xenograft, KUCaP, which was established from a patient treated with antiandrogen bicalutamide.

Pharmacokinetics, Antitumor Activity, and Safety of ODM-201 in Patients with Chemotherapy-naive Metastatic Castration-resistant Prostate Cancer: An Open-label Phase 1 Study.

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Subjects responses were compared to an age-matched comparison group of women, although no further details about how this comparison group were provided.

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Also called ABR test, auditory brain stem response test, and brain stem auditory evoked response test.

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However, given that the observed percentage increase in total testosterone levels following tamoxifen therapy was several-fold higher than the increase in serum SHBG levels, a clinically relevant impact on sexual function is unlikely in this trial.

Project and national patterns of prostate cancer detection and treatment.

CT scan of the region to be treated is done.

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As a minimum requirement, include a disease-specific history, DRE, serum PSA, haemoglobin, serum creatinine and alkaline phosphatase measurements in the diagnostic work-up.

The goal of androgen ablation is to identify those agents that will most consistently achieve and maintain the lowest testosterone levels possible.

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It is unclear whether there is any difference in efficacy between these variants, except from the fact that about 10% more of sodium vaproate is needed than valproic acid without sodium to compensate for the sodium itself.

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Journal of the American Medical Association study cited above) included men who did not undergo further treatment after biochemical recurrence occurred.

Nilandron (nilutamide) and Xtandi (enzalutamide) are antiandrogens that function similarly to bicalutamide.

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H874Y mutant AR in CWR22 xenograft did not significantly affect in vivo steroid specificity and concluded that an in vivo model is essential for determining the effect of an AR mutation.

Early versus delayed hormonal therapy for prostate specific antigen only recurrence of prostate cancer after radical prostatectomy.

DHEA (from intrinsic or circulating sources depending on the tissue) is subsequently acted on by HSD3B to form AED and HSD17B3 (or AKR1C3) to form testosterone which is converted to 5α-DHT via SRD5A.

Whitney U-test was used for statistical comparison of median PSA changes.

HRQL in men after treatment for localized prostate cancer.

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Step (II) is a solution of bicalutamide in a mixed solvent of ethyl acetate and heptane.

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The solution is concentrated to generally 2 ml-10 ml, preferably 2 ml-6 ml, per 1 g of bicalutamide (under atmospheric pressure to reduced pressure).

The significance of this hypothesis, however, is subject to a number of important questions.

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At present, the primary approaches for the initial hormonal management of prostate cancer to reduce circulating serum levels of testosterone are estrogens, surgical orchiectomy, LHRH hormone agonists, and LHRH antagonists.

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